Lacunar infarction

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[edit] Discussion of Lacunar infarction

  • Lacunar infarctions are small infarctions lying in deeper noncortical parts of the cerebrum and brainstem.
  • Caused by occlusion of penetrating branches that arise from the middle cerebral, posterior cerebral, and basilar arteries and less commonly from the anterior cerebral and vertebral arteries.
  • Sites of predilection are the basal ganglia, thalamus, internal and external capsule, ventral pons, and periventricular white matter.
  • Lacunar infarctions can be differentiated from Virchow-Robin spaces by signal intensity characteristics.

[edit] Imaging Findings for Lacunar infarction

  • An acute lacunar infarction (12 hours up to 7 days) appears as a small high-signal-intensity region on T2-weighted and FLAIR images and as a hypointense area on T1-weighted images.
    • High signal intensity is seen on diffusion-weighted images with corresponding low signal intensity on the apparent diffusion coefficient map.
    • Enhancement is variable
  • A chronic lacunar infarction is better defined and has high signal intensity on T2-weighted images and low signal intensity on T1-weighted images. On FLAIR images, a hyperintense lesion or a lesion with a hypointense center and a hyperintense rim reflecting gliosis is seen.
    • Enhancement may last for up to 8 weeks after the acute event

[edit] Images

Patient #1: Chronic lacunar infarctions on CT

[edit] See Also

[edit] External Links

[edit] References for Lacunar infarction